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ES 34 Ch. 23 This chapter discusses the different etiologies of gingival enlargement. Gingival enlargement can be due to the inflammatory process, side effects of drugs, systemic conditions, or gingival tumors. It is important to recognize the different etiologies of gingival enlargement so that it could be treated accordingly. Ch. 24 NUG, primary herpetic gingivostomatitis, and pericoronitis are all classified as acute gingival infections. NUG can be diagnosed based on clinical findings because the bacterium associated with NUG is not definitive of the infection. Herpetcic gingivostomatitis can be diagnosed both clinically and also by a viral culture. Periocoronitis can be diagnosed based on clinical observations.

Ch 23. Gingival enlargement is due to either etiologic factors or pathologic. Gingival inlargement for example is caused from inflammation, drug induced, systemic diseases, certain conditions such as prgnancy, malnutrition, and puberty, tumors, and some are idiopathic. Ch. 24 Acute gingival infections include NUG, primary herpetic gingivostomatitis and pericornitits which all can be identified by clinical observation.

Ch. 23: The most common form of gingival enlargement is due to plaque residing in proximity to the gingiva, causing inflammatory enlargement. This shows characteristic signs of gingivitis such as erythema, edema, and increase in size. Many times this is seen on the interdental papilla especially. There are other causes for gingival enlargement such as medications, idiopathic causes which may be genetic, systemic disease, or neoplasms. Ch. 24: Acute gingival infections include NUG, primary herpetic gingivostomatitis, and pericoronitis. These are caused by specific bacterial (or in the case of herpetic gingivostomatitis, viral) pathogens. These infections are able to be identified based on their clinical features.

Ch 23: Gingival enlargement can result due to several different factors such as: drug related, systemic conditions, and tumor related factors. Gingival enlargement can also be determined depending on the location as well. The enlargement can result from changes in the tissue that are either acute or chronic; chronic being the most common form. Acute inflammation may occur due to trauma while chronic may be due to plaque that has been present for a long period of time. Ch 24: NUG, herpetic gingivostomatitis, and pericornitis are all forms of acute gingival infections. These infections can be identified by their distinct clinical features.

The Big ideas for chapter 23 is the gingiva can increase in size depending on the etiology factors and pathological changes in the disease process. Gingival enlargement may result from either chroni or acute inflammatory changes. Chronic is mainly associated with the prolonged exposure of plaque and acute are abcess that are localized and painful. Medication used such as anticonvulsants, calcium channel blocker and immuno suppressants are also know to cause gingival enlargements.

Chapter 24 talks about the different clinical featurea associated in acute gingival infections these include NUG, gingivostomatitis and periconitis. All result in the impared host response that categorizes these specific disease to become acute gingival infections.

Chapter 23- Gingival enlargement can occur do to etiologic factors or pathologic changes. Chapter 24- The clinical appearance and etiology of necrotizing ulcerative gingivitis, primary herpetic gingivostomatitis, and pericoronitis.

CH23. The primary cause for gingival enlargment is plaque related, however that is not the only cause of gingival enlargment. Gingival enlargment can also be caused my certain medications and systemic involvement. In it important to be aware of the etiology in order to treat the patient appropriatly. CH.24. NUG is an acute inflammation of the gingiva from a certain species of pathogenic bacteria. A Through Patient history, as well as clinical appears can help differentiate NUG from gingivitis. -- CH 23 gingival enlargement is a common feature of gingival diseases. There are many etiologies of gingival enlargement including inflammatory, drug induced, assoc. w/conditions, systemic diseases and neoplastic enlargement. Enlargement can be localized, generalized, marginal and papillary. Ch 24 acute gingival infections include NUG, primary herpetic gingivostomatitis, and pericoronitis and they all have distinct clinical appearances.

Ch.23- The big idea of this chapter is gingival enlargement and the different etiologies that can cause it such as medications, sytemic conditions such as leukemia, plaque-acute or chronic enlargment, pregnancy, puberty, and even mouth breathing. Ch.24- The big idea of this chapter is acute gingival infections such as NUG, primary herpetic gingivostomatitis, and pericoronitis, and includes the different etiologies, clinical features, and epidemiologies and prevalence. AS36

Chapter 23: Discusses the possible causes of gingival enlargement including drug-induced, idiopathic causes, associated with systemic diseases, gingival tumors, and enlargement due to inflammation. Chapter 24: Discusses the clinical features, diagnosis and etiology of NUG, NUP, and primary herpetic gingivostomatitis, and pericoronitis

Chapter 23: talks about the different types of gingival enlargment and how they can also be associated with systemic diseases.It talks about the clinical features with each type and It also talks about false enlargment. Chapter 24: This chapter talks about NUG and discusses oral signs, clinical features, as well as the diagnosis. It also talks about primary herpetic gingivostomatitis and the oral signs and symptoms, as well as the diagnosis. SF25

Chp 23: Gingival enlargement can be a clinical feature caused by many factors; systemic disorders, neoplasms, plaque biofilm accumulation with pathogens eliciting the inflammatory response, and medication side effect. Chp. 24: The three acute gingival infections discussed; NUP, primary herpetic gingivostomatitis, and pericoronitis have one common feature: pain. All three are very uncomfortable for the patient. Pericoronitis has varying degrees of inflammation but has the potential to involve the pericoronal flap covering the occlusal surface of an adjacent tooth, preventing the patient not to bite down all the way. Primary herpetic gingivostomatitis has an acute onset and is most observed in children 1-6 years, but is also seen in adolescents and adults. It is an infection of the oral cavity caused by the herpes simplex virus that may lay dormant in adulthood and may be triggered by various stimuli such as trauma or stress. Ulcerative lesions may persist for 7-10 days and cause soreness of the oral cavity which can interfere with eating and drinking. NUG has an acute onset and is associated with an impaired host response. NUG is characterized by painful gingival tissue that presents with punched-out papilla and a gray pseudomemranous sloughing of the gingival tissue. NUG can cause tissue destruction involving the periodontal attachment apparatus if not treated (long standing disease or severe immunosuppression) and when bone loss does occur, it then is called NUP. ACS #35