Week+10+Big+Ideas

What were the Big Ideas in the reading and class discussion in week #10? Ch 28. In chapter 28, the author explains the different factors that lead to bone destruction including gingival inflammation that extends into the bone; distraction caused by trauma from occlusion, and systemic disorders. Pattern of the bone loss include horizontal, vertical, bone deformities, craters in the osseous, ledges, and furcation involvement. Ch 32. In chapter 32, the author explains the etiology, clinical and microscopic findings of necrotizing ulcerative periodontitis Ch 33. In chapter 33, the author explains the clinical and radiographic characteristics associated with localized and generalized aggressive periodontitis; the age of onset of the disease; risk factors; and predisposing factors. Ch. 28-Most common cause of bone loss is a result of periodontal disease, which occur form gingival inflammation extending into the supporting periodontal tissues. Destruction results in loss of collagen and alveolar bone with deepening of the periodontal pocket. Bone destruction can also be a result of trauma from occlusion that can occur with or without inflammation.

Ch. 32- Necrotizing ulcerative periodontitis (NUP) leads to periodontal attachment and bone loss. The disease has a sudden onset, and presents with ulceration and necrosis of the interdental papilla, also referred to as punched out papilla, and there is a bad odor. The condition is painful and the lesions bleed easily. The condition most commonly presents in individuals that immune system is compromised.

Chapter 28- The destruction of bone caused by trauma, systemic disorders, and periodontal disease. Chapter 32- The clinical appearance and etiology of necrotizing ulcerative periodontitis. Chapter 33- The clinical appearance, radiographic appearance, the occurrence, and risk factors for aggressive periodontitis.

Ch. 28 Bone destruction is caused by gingival inflammation which is caused from extensions of inflammation from the marginal gingiva, trauma from occlusion from increased tension and compression of the periodontal ligaments, and systemic disorders. The anatomical features affect the bone destructive pattern in periodontal disease. Bone destruction includes horizontal which is the most common and vertical bone loss that occurs in an oblique direction, osseous craters, bone contours and furcations. Ch. 32The clinical features, charatcteristics and etiology of Necrotising Ulcerative Periodontitis. Clinical features include "punched out papilla". Characteristics include AID/HIV patients and the eitiology includes immunocompromised patients, physical stress and malnutrition.Ch. 33Agressive periodontits includes locolized and generalized. Localized usually has an age onset at about pubery and invoves the A.a bacteria and it usually affects localized areas of the first molars and incisors causing a verticle bone defect. Generalized agressive periodontits usually affects those under age 30 and is severe bone loss afecting the majority of the teeth. The risk factors include microbiologic factors such as the presence of A.a. Also immunologic factors such as functional defects of PMNs and monocytes. Genetic factors can contribute suggesting that it may be inherited as well environmental factors like smoking.

CH 28: Bone loss is a result of several different factors which include systemic conditions, trauma in relation to occlusion, or from and extension of gingival inflammation. There are different classifications of bone loss such as; horizontal which is the most common and vertical which usually results from aggressive bone loss. CH 32: NUP and NUG share some similarities, but what separates them from one another is that NUP causes destruction of attachment and bone loss. NUP is more than likely to be seen in patients who are immune compromised such as in patients with AIDS. CH 33: There are three forms of aggressive periodontitis; localized, generalized, and rapidly progressive periodontitis. These forms of periodontitis all affect the periodontium in a different manner. It is important that we are able to recognize and understand the different features for each different type so that we can correctly identify it and treat it.

Ch. 28- Bone loss in periodontal disease is caused and influenced by several factors, one of the main causative factors is plaque and the destruction of host periodontal tissues. In periodontal disease, there can be horizontal bone loss, which is the most common form, and vertical bone loss. There can also be furcations, osseous craters, and other types of bone defects.Ch. 32- NUP is a type of periodontitis that resembles NUG with the exception that in NUP, there is bone loss present. Although becteria are implicated as a key factor in its development, NUP (often an extension of NUG) can also be influenced by poor oral hygiene practices, preexisting periodontal disease, smoking, infections, stress, malnutrition, and immunecompromised status.Ch. 33- Aggressive periodontitis can be localized or generalized. In localized aggressive (LAP), onset usually occurs at puberty and in generalized (GAP), it usually occurs in indivduals under 30 years of age. The rate of bone loss is thought to be 3-4 times faster than chronic periodontitis. A.a and P. gingivalis seem to the main bacteria implicated in cases of LAP and GAP.

Ch.28 The big idea for this chapter is the bone destruction in periodontal disease that is caused by the extension of inflammation which is associated with by the changes in composition of bacterial plaque. Bone destruction occurs in episode of rapid destruction followed by inactivity of destruction. Both bacterial plaque and the host response are factors that activate the degree of bone destruction. Trauma from occlusion is the increase of compression and tension of the periodontal ligaments is another cause of periodontal destruction. Ch.32 Talks about NUP and the differentiation of nug and nup. Nup is categorized as very destructive progression in the periodontal attachment and bone loss. Basically the necrosis and ulcerations of the interdental papillae and the gingival margin clinically looks bright red painful and bleeds excessively. CH.33 the two types of aggressive periodontitis localized and generalized. LAP onset is about puberty affecting the first molars and incisors with an attachment loss of two permanent teeth. With GAP mainly affects young adults under 30 years of age and minimal amount of plaque. Risk factor associated with aggressive periodontitis is the microbiologic factor of increased amounts of A.A, immunological factors, genetic and environmental those include smoking which increases the extent of destruction.

Ch. 28 This chapter discusses how bone loss can occur and the patterns of bone loss. The most common cause of bone loss is due to untreated gingival inflammation. The cause of the bone loss due to the extension of gingival inflammation is the microorganisms that are associated with periodontitis and also the host response. Other causes of bone loss are bone loss due to trauma and bone loss due to systemic disorders. The most common pattern of bone loss is horizontal bone loss. There is also vertical bone loss, craters, ledges, and furcation involvement. Ch. 32 NUP is characterized by the necrosis of the interdental papilla and the gingival margin. There is often a foul odor associated with it. The difference between NUP and NUG is NUP is associated with attachment and bone loss. The progression of NUP is linked to impaired immune response and a lowered host resistance. Ch. 33 Aggressive periodontitis often affects young people under the age of 30. Age can be used to distinguish aggressive periodontitis from chronic. Aggressive periodontisis can be both localized and generalzed. Localized aggressive perio. presents with localized attachment loss on two permanent teeth, one being a molar, and involving no more than two teeth other than first molars and incicors. Generalized aggressive perio. presents with generalized interproximal attachment loss of at least three permanent teeth other than molars and incisors. (ES 34) Ch.28Big idea is about bone loss and its destruction pattern and how the inflammation diease contributes to bone destruction. It also reffers to the differnt type of bone destuction caused by trauma and systemic issues. This chapter also talks about the different desruction patterns such as horizontal, vertical and osseous deformities.

Ch.32NUP is the big idea for ch 32. It goes into all aspects of NUP such as how it looks clinically and what microorganisims are in it microscopicly. The classical features of the punched out pappilla and the mass bacteria that accopanies this type of gingivitis. Ch. 33Is about agressive perio and the clinical characteristics and what age it usually presents. It breaks down the periodontal destruction pattern and the bacteria associated with it. It also touches on what the diiferent factors that can cause aggressive perio such as immunologic, genetics and enviromental.

Ch. 28 The big idea in chapter 28 is the destruction patter of bone loss in periodontal disease. The most common cause of bone loss is due to acute gingivitis, the inflammation process and pathogenic bacteria. There are different patterns of destruction in periodontal disease. Horizontal is the most common patter of bone loss, however vertical and furcation involvement is also seen in radiographs. Periodontal disease can be be associated with systemic issues and trauma (although not as common).

Ch. 32 NUP stand for Necrotizing Ulcerative Periodontitist and effects the gingival tissue that spreads into the periodontium and continues destruction. NUP is painful and the patient may also complain of a metallic taste. The clinician will noticed classic signs of NUP such as a punched out pippalla. A bacterial culture would identify specific species of bacteria in relation to the host defense.

Chapter 28: This chapter talks about different types of bone loss and destruction and the different kinds of defects that can happen during periodontal disease, as well as deformities that can occur. Chapter 32: This chapter talks about NUP and gives the characteristics and features of it, the microscopic findings, and the etiology of it. Chapter 33: This chapter talks about localized aggressive periodontitis and localized aggressive periodontitis and discusses the clinical characteristics of each as well as risk factors for both of them. SF 25 --- Ch 28 destruction of alveolar bone caused by several factors such as an extension of gingival inflammation, trauma, systemic disorders. Bone destruction patterns ex. Horizontal and vertical boneloss. Ch 32 Necrotizing ulcerative periodontitis. May be extension of NUG, immunocompromised individuals are more likely to suffer from this disease especially HIV/AIDS pts. Ch 33 aggressive perio. Localized first molar/incisor affected. High levels of A.a. usually affects younger individuals (under 30). May be familial pattern, Smoking may be a risk factor.

Chapter 28: Bone destruction can be caused by gingival inflammation, trauma from occlusion, or systemic disorders. There are many factors that determine morphology in periodontal disease such as lipping, food impaction, aggressive forms of periodontitis and exostoses.

Chapter 32: NUP is distinguished from NUG because NUP has bone loss. Patients that are susceptible to NUG are usually immunocompromised in some way. Patients with HIV/AIDS are an example of an immunocompromised host. NUG can rapidly progress to NUP if a patient smokes, has a viral infection, under stress, or malnutrition.

Chapter 33: The two forms of aggressive periodontitis are localized and generalized periodontitis. In localized usually has an age of onset by puberty and the periodontitis is localized in the at least one molar and no more than two incisors. Generalized periodontitis usually affects individuals under the age of 30 and affects 3 other teeth besides the molars and incisors.

Ch28- The big idea is the patterns of bone loss, which can be caused by gingival inflammation and malocclusion. Also some factors affecting bone loss are systemic disorders and bone morphology. Ch32- The big idea is NUP which is like NUG but with bone loss. It can be casued by stress, from a compromised immune system, if you have HIV, or malnutrition. Ch33- Th ebig idea is aggressive periodontitis which is characterized by vertical bone loss. It can be generalized or localized. When its localized it usually affects the first molar/incisor. There are high levels of A.a. and usually affects younger individuals (under 30). AS36

Chp. 28: Bone loss can be caused by an extension of gingival inflammation, invasion of pathogens and the ensueing bone loss marks the transition from gingivitis to periodontitis. Bone loss occurs in episodic periods of destruction, factors involved are bacterial and host mediated. Other causes of bone loss can be from trauma from occlusion, systemic disorders and variations in bone morphology. Patterns of bone loss in periodontal disease can include horizontal (most common), vertical and bone deformities. Chp. 32: NUP represents an acute form of periodontal destruction that corrolates with host defenses compromised in some form. NUP is categorized as a necrotic disease that affects deeper tissues of the periodontium, resulting in loss of connective tissue attachment (periodontal ligament) and alveolor bone. Clinical features of NUP include interdental gingival necrosis (ulcerations with whitish yellow soft pseudomembranous lesions surrounded by erythematous halo’s), pain, and bleeding, blunted and cratering of papillae, and fetid breath; variable features include lymphadenopathy, fever, and malaise. Could be progression from untreated NUG. Bacterial plaque is the predisposing factor for NUP and is usually complicated by the presence of secondary factors such as acute psychologic stress, tobacco smoking, poor nutrition, all of which can contribute to immunosuppression (systemic conditions, e.g., HIV infection.) Chp. 33: Aggressive periodontitis is characterized by periodontal destruction that becomes clinically significant during adolescence or early adulthood, disease progression is rapid. Classified by localized and generalized types. Localized is characterized by bone loss around the first molars and incisors involving interproximal attachment loss on at least two permanent teeth, one is the first molar, and involving no more than two teeth other than first molars and incisors. A major clinical feature is the lack of clinical inflammation despite the presence of deep pockets and advanced bone loss and a minimal amount of plaque. Generalized is characterized by a more widespread pattern of periodontal destruction, affecting at least three teeth other than the first molars and incisors. Occurs in episodes with rapid periods of destruction followed by a latent period. ACS #35